Furthermore, the Ex4a(+)WT1 isoform and complete WT1 isoforms which includes equally Ex4a(+) and main WT1 isoforms were being quantified by quantitative real-time PCR (Fig 6C). The results verified that Ex4a(+)WT1 isoform was greater and significant WT1 isoforms had been diminished by apoptosis. When etoposide was utilized as an apoptosis inducer, the related benefits had been received (data not s
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